Completed molecular model for the Alzheimer's disease amyloid hypothesis reveals a new role for the metabotropic glutamate receptor 5 as the link between the prion protein and the FYN cellular messenger
Schematic of a normal neuronal microtubule (top) and a microtubule no longer stabilized by tau proteins (Image: Resident Mario via Wikipedia)
Artist's conception of normal and tangled tau proteins in the microtubules of neurons (Photo: National Institute on Aging)
PET scans of beta amyloid in a normal brain at left, and in the brain of a patient with Alzheimer's at right (Photo: National Institute on Aging)
Formation of beta amyloid plaques that interfere with communication paths between neurons in the brain of an Alzheimer's patient (Photo: National Institute on Aging)
New research has provided "strong hope" of finding a drug to combat Alzheimer’s (Image: Shutterstock)
A team of researchers at Yale University has completed a molecular model for Alzheimer's disease by identifying a protein that plays a key role in its onset. Promisingly, the study showed that when the activity of this protein is blocked by an existing drug, mice engineered as models for human AD recover their memories.
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